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Antiabortion-Rights Advocate To Oversee HHS Office Of Faith-Based And Community Partnerships
President Obama has named Alexia Kelley -- Catholics in Alliance for the Common Good co-founder and former executive director -- to the position of director of Faith-Based and Community Partnerships at HHS, Salon"s "War Room" reports. According to Salon"s the "War Room," CACG primarily has worked to find ways to reduce the demand for abortion procedures rather than advocate for laws to restrict access. However, CACG"s Web site reads, "Catholics in Alliance believes in the sanctity of all human life -- from conception until natural death" (Madden, "War Room," Salon, 6/4).Catholics for Choice President Jon O"Brien writes in The Hill"s "Congress Blog" that the appointment is "a defeat for reason and logic" and "calls into question whether President Obama"s administration is serious about reducing the need for abortion." According to O"Brien, Kelley "is on record with her support for restrictions on access to abortion," although CACG has sought to "avoid the question of legalization at every turn." O"Brien continues that the group also used "flawed economic data to support anti-poverty measures as a means to reduce the number of abortions," and "opposed evidence-based prevention methods such as contraception and comprehensive sexuality education" (O"Brien, "Congress Blog," The Hill, 6/4).Officials at the White House and HHS did not return calls for comment. CACG spokesperson Jennifer Goff said, "Catholics in Alliance for the Common Good is working toward reaching common ground in order to make real progress on the moral and political challenges our country faces instead of resorting to spurious attacks launched by those who are more concerned with inflaming the culture wars than effecting positive change" ("War Room," Salon, 6/4).
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Mouse Model Of Parkinson's Reproduces Nonmotor Symptoms
The classic symptoms of Parkinson"s disease involve tremor, stiffness and slow movements. Over the last decade, neurologists have been paying greater attention to non-motor symptoms, such as digestive and sleep problems, loss of sense of smell and depression.
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Reprogramming Human Cells Without Inserting Genes
A research team comprised of faculty at Worcester Polytechnic Institute"s (WPI) Life Sciences and Bioengineering Center (LSBC) and investigators at CellThera, a private company also located at the LSBC, has discovered a novel way to turn on stem cell genes in human fibroblasts (skin cells) without the risks associated with inserting extra genes or using viruses. This discovery opens a new avenue for reprogramming cells that could eventually lead to treatments for a range of human diseases and traumatic injuries by coaxing a patient"s own cells to repair and regenerate the damaged tissues.
Medical Devices

New Johns Hopkins Study Betters The Odds Of Success In Predicting The Return Of Prostate Cancer

Cancer experts at Johns Hopkins say a study tracking 774 prostate cancer patients for a median of eight years has shown that a three-way combination of measurements has the best chance yet of predicting disease metastasis. The new prediction method comprises the length of time it takes for PSA (prostate-specific antigen) to double, Gleason score (a numeric indicator of prostate cancer aggressiveness as seen under the microscope), and the interval between surgical removal of the prostate and the first detectable PSA level. According to Johns Hopkins investigators, combining these three measurements more accurately estimates risk that the cancer has spread than do other methods and should help determine which patients may benefit from additional therapy when PSA levels rise after surgery to remove the prostate. Findings from the study presented at the June 2009 annual meeting of the American Society of Clinical Oncology (ASCO) may also help resolve the debate on when and in what form secondary treatments should occur. "There is much debate on whether to prescribe immediate treatment for a man whose PSA begins to rise after he has had prostate cancer surgery, or to delay it," says Emmanuel Antonarakis, M.D., Johns Hopkins Kimmel Cancer Center investigator. "Studies suggest that most men live the same length of time overall whether they receive therapy at the first sign of a rising PSA or wait until the cancer has spread to other sites." After reviewing the records of 774 men whose PSA rose after surgery to remove the prostate, the researchers found that Gleason score and two measurements for PSA were the strongest risk factors for prostate cancer metastasis. Men with Gleason scores in the highest range, between eight and 10, were twice as likely to develop metastatic cancer. In men whose PSA became detectable within three years after surgery, cancer was more than three times more likely to spread to other organs. Finally, men whose PSA doubled the fastest, within three months, were more than 20 times more likely to develop metastatic cancer than men whose PSA look longer than 15 months to double. For men enrolled in the study, it took a median of 10 years for the disease to reappear on imaging scans. "The 10-year average will not apply to every man, so we wanted to know what factors put men at higher risk for their cancer progressing earlier," says Mario Eisenberger, M.D., professor of oncology at the Johns Hopkins Kimmel Cancer Center. An increase in PSA, or prostate specific antigen, occurs in approximately 20 percent to 30 percent of men after surgery to remove the cancerous prostate, says Antonarakis. In these patients, the newly emerging prostate cancer cells are rarely detectable on imaging scans. When faced with the likelihood that their cancer has spread, many men opt to undergo hormone therapy, which blocks testosterone production, a fuel for prostate cancer. The side effects, which mimic those of menopausal women, include hot flashes, night sweats, osteoporosis, metabolic syndrome and coronary disease, and can be debilitating, says Antonarakis. Besides immediate hormone therapy, other options for men whose PSA is rising are to use hormone therapy intermittently, enroll in clinical trials testing experimental therapies or combinations of them, or to "watch and wait" until imaging scans can locate metastatic disease. Notes: Data on the prostate cancer patients involved in this study were collected from a database maintained by Patrick C. Walsh, M.D., at the Johns Hopkins Brady Urological Institute. The information spans nearly 20 years of patient records at Johns Hopkins. The research was funded by the National Cancer Institute, the Prostate Cancer Foundation, and the Department of Defense Prostate Cancer Research Program. In addition to Antonarakis and Eisenberger, the following researchers participated in the study: Bruce Trock, Zhaoyong Feng, Elizabeth Humphreys, Michael Carducci, Alan Partin, and Patrick Walsh from Johns Hopkins. Amy Mone Johns Hopkins Medical Institutions


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