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Explaining Oral/Body Inflammatory Connection
Is your head where your heart is? It may be now. A strong connection between periodontal disease and cardiovascular disease (CVD) has been suggested in recent clinical studies. As many as 75 percent of adults in the United States have been affected by periodontal disease and an estimated 80.7 million adults (1 out of every 3) have been a victim of CVD in 2006 according to the American Heart Association. From the 80.7 million adults in the United States, 38.2 million are less than 60 years of age, which is almost 50 percent. According to Marvin J. Slepian, MD, and Neil R. Gottehrer, DDS, who is lead a discussion titled "Oral Body Inflammation Connection" during the 57th Annual Meeting of the Academy of General Dentistry (AGD), these findings strengthen their belief that oral infections contribute to CVD morbidity and connection of chronic infections and CVD. The AGD"s Annual Meeting is take place in Baltimore, MD, July 8-12, 2009.
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Deep Brain Stimulation For Depression Pilot Study Demonstrates Sustained Improvement In Depression Symptoms
According to the latest data in a clinical study supported by St. Jude Medical, Inc. (NYSE:STJ), deep brain stimulation (DBS) therapy for depression may provide sustainable improvement in depression symptoms among patients with major depressive disorder. Study results will be presented at the American Psychiatric Association (APA) meeting in San Francisco.
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NFL Players Have More Favorable Glucose Levels, Similar Cholesterol Levels, Higher Rate Of High Blood Pressure Compared To Other Healthy Young Men
Despite being larger in size and heavier in weight, an analysis of the cardiovascular disease risk factors of about 500 National Football League players finds that overall, they have a similar cardiovascular risk profile compared to the general population. The NFL population was found to have a lower incidence of impaired fasting glucose and similar prevalence of abnormal cholesterol and triglyceride levels as compared to a sample of healthy young-adult men, but have an increased prevalence of high blood pressure, according to a study in the May 27 issue of JAMA.
Endocrinology

Scientists Locate Disease Switches

New perspectives in the treatment of disease The team, led by Professor Matthias Mann of Novo Nordisk Center for Protein Research at the University of Copenhagen and the Max Planck Institute for Biochemistry in Germany, have detected 3,600 acetylation switches in 1,750 different proteins. These switches, which regulate protein functions, may prove to be a crucial factor in human ageing and the onset and treatment of diseases such as cancer, Alzheimer"s disease and Parkinson"s disease. The results of the team"s work have been published in the current edition of the journal Science. "This is more than just a technological achievement, it has also expanded the number of known acetylation switches by a factor of six, and it gives us for the first time a comprehensive insight into this type of protein modification," says Professor Mann. A given protein can perform more than one task, and how it behaves is regulated by adding a small molecule that acts as a "switch" which can turn on the different tasks. Acetylation is essential for cells" ability to function normally. Defective protein regulation plays a role in ageing and the development of diseases such as cancer, Parkinson"s and Alzheimer"s. "With the new mapping, we can now begin to study and describe how acetylation switches respond to medications that could repair the defects on them. It can have a major impact on medical care," says Professor Mann, adding that medications to repair the damaged protein regulation are already showing promising in the treatment of cancer. Cooperating proteins The team also discovered that acetylation modification occurs primarily on proteins that work together, and that these switches have much greater consequences for the organism"s function than previously thought. In one example, the function of Cdc28, an important growth protein in yeast, can be disrupted by the addition of an acetylation button, ultimately affecting the organism"s ability to survive. The results of the team"s research were published in the 17 July 2009 edition of Science. University of Copenhagen


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